Recently, in a research report published in the international journal Nature Microbiology, researchers from institutions such as York University have elucidated the working mechanism of hepatitis B virus, or provided new hope for later researchers to develop new therapies for the treatment of hepatitis B.
Today, hepatitis B is a major cause of liver cancer in the global population. In the article, researchers identified a special “assembly code†from the genetic material of the hepatitis B virus. This code can help the virus produce a protective cover. The creation of more infective virus particles provides the basis; researchers have pointed out that the signals generated by RNA can help viral proteins overcome "engineering problems," thereby helping to assemble viral proteins into special geometric patterns. Prof. Reidun Twarock said that this is a bit like a chain on a bicycle. If we can't attach the chain to the sprocket, it will become chaotic and lose its function. Once it is assembled correctly, the chain structure will place the pedals on the same wheel. Connected to work better.
Now researchers have found the same process in the RNA signals of hepatitis B virus and viral proteins. The viral proteins can be attracted by these signals, and then promote the assembly of viral proteins into precise molecular machines, thereby promoting the virus to produce genetic material copies and enhance The infectivity of the virus. Hepatitis B virus can be transmitted through blood and body fluids. Currently, more than 2 billion people in the world have been infected with the virus, and today there are still 350 million hepatitis B virus carriers.
Hepatitis B virus infection often occurs in Europe, but it is very common in parts of Africa and Asia, especially in China. At present, more than one million Americans are infected with hepatitis B virus. The corresponding therapy includes expensive drugs and even liver. transplant. Researcher Peter Stockley believes that hepatitis B vaccine can protect the body against viral infections, but it cannot be cured once the virus is infected. Although some drugs can reduce the symptoms of patients, they cannot change the long-term status of patients.
Since HBV is also transmitted from person to person, researchers often compare the virus with HIV. Unlike HIV, there is currently no effective drug to improve the quality of life of patients with hepatitis B. Now researchers have clarified that The molecular mechanism of virus assembly, they may later be able to interfere with the mechanism of interaction between viral and RNA signals. Researchers are now working with researchers at the National Institutes of Health to identify potential drug candidates. They hope that in the future, drugs that disrupt the link between RNA and viral proteins can be found, effectively suppressing viral replication.
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